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With research published in the best periodics, our researchers have effectively collaborated with the development of the biological sciences.
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Agenda IB
Luís Felipe de Toledo Ramos Pereira
Amphibians - Conservation, Taxonomy, Natural History, Behavior, Bioacoustics, Diseases
Marcelo Alves da Silva Mori
The main purpose of our lab is to identify and characterize molecular mechanisms involved in the aging process and in response to dietary interventions. We pay particular interest to understand how metabolism contributes to the genesis of age-related diseases like type 2 diabetes, cardiovascular diseases, and neurodegenerative diseases. We focus on the fat tissue - an important site of metabolic integration in multicellular organisms - and ask ourselves how this tissue responds to changes in energy balance and signals to other tissues to inform them about these changes. If not adjusted, this mechanism leads to several chronic diseases, especially in middle aged and elderly individuals. We identified that this control is at least in part exerted by changes in microRNA biogenesis in adipose tissue. When regulated, this process affects animal susceptibility to oxidative stress and influences lifespan.
Sílvio Roberto Consonni
The main purpose of our lab is to recognize the effects of multiparity and ageing in cells and extracellular matrix during postpartum recovery at birth canal (pubic symphysis, uterine cervix and vagina) in mice. We pay particular interest to understand how birth and ageing have a significant effect in tissue homeostasis, which could help us to understand the pathophysiology of pelvic organ prolapse, urinary incontinence and preterm. We focus on the connective tissue - an important site of cellular, molecular and hormonal signaling - and ask how this tissue responds (or not) to changes during and after first and multiple pregnancies. Using morphological, biochemical and molecular assays, we identified that recovery processes are regulated by cells and molecules in the birth canal and multiparity and ageing led them to not recover in the reproductive matrix mice.